Zhu ZI, Clancy CE. (2007). L-type Ca2+ channel mutations and T-wave alternans: a model study. American journal of physiology. Heart and circulatory physiology. 293 [PubMed]

See more from authors: Zhu ZI · Clancy CE

References and models cited by this paper

Alseikhan BA, DeMaria CD, Colecraft HM, Yue DT. (2002). Engineered calmodulins reveal the unexpected eminence of Ca2+ channel inactivation in controlling heart excitation. Proceedings of the National Academy of Sciences of the United States of America. 99 [PubMed]

Berrou L, Bernatchez G, Parent L. (2001). Molecular determinants of inactivation within the I-II linker of alpha1E (CaV2.3) calcium channels. Biophysical journal. 80 [PubMed]

Bondarenko VE, Szigeti GP, Bett GC, Kim SJ, Rasmusson RL. (2004). Computer model of action potential of mouse ventricular myocytes. American journal of physiology. Heart and circulatory physiology. 287 [PubMed]

Branchaw JL, Banks MI, Jackson MB. (1997). Ca2+- and voltage-dependent inactivation of Ca2+ channels in nerve terminals of the neurohypophysis. The Journal of neuroscience : the official journal of the Society for Neuroscience. 17 [PubMed]

Chinushi M, Restivo M, Caref EB, El-Sherif N. (1998). Electrophysiological basis of arrhythmogenicity of QT/T alternans in the long-QT syndrome: tridimensional analysis of the kinetics of cardiac repolarization. Circulation research. 83 [PubMed]

DeMaria CD, Soong TW, Alseikhan BA, Alvania RS, Yue DT. (2001). Calmodulin bifurcates the local Ca2+ signal that modulates P/Q-type Ca2+ channels. Nature. 411 [PubMed]

Echebarria B, Karma A. (2002). Instability and spatiotemporal dynamics of alternans in paced cardiac tissue. Physical review letters. 88 [PubMed]

El Habbal MH, Mahoney CO. (2002). QT interval in children with sensory neural hearing loss. Pacing and clinical electrophysiology : PACE. 25 [PubMed]

Faber GM, Silva J, Livshitz L, Rudy Y. (2007). Kinetic properties of the cardiac L-type Ca2+ channel and its role in myocyte electrophysiology: a theoretical investigation. Biophysical journal. 92 [PubMed]

Fabritz L et al. (2003). Effect of pacing and mexiletine on dispersion of repolarisation and arrhythmias in DeltaKPQ SCN5A (long QT3) mice. Cardiovascular research. 57 [PubMed]

Findlay I. (2004). Physiological modulation of inactivation in L-type Ca2+ channels: one switch. The Journal of physiology. 554 [PubMed]

Fox JJ, McHarg JL, Gilmour RF. (2002). Ionic mechanism of electrical alternans. American journal of physiology. Heart and circulatory physiology. 282 [PubMed]

Fox JJ, Riccio ML, Hua F, Bodenschatz E, Gilmour RF. (2002). Spatiotemporal transition to conduction block in canine ventricle. Circulation research. 90 [PubMed]

Hofmann F, Lacinová L, Klugbauer N. (1999). Voltage-dependent calcium channels: from structure to function. Reviews of physiology, biochemistry and pharmacology. 139 [PubMed]

Hudmon A et al. (2005). CaMKII tethers to L-type Ca2+ channels, establishing a local and dedicated integrator of Ca2+ signals for facilitation. The Journal of cell biology. 171 [PubMed]

Imredy JP, Yue DT. (1994). Mechanism of Ca(2+)-sensitive inactivation of L-type Ca2+ channels. Neuron. 12 [PubMed]

Karma A. (1993). Spiral breakup in model equations of action potential propagation in cardiac tissue. Physical review letters. 71 [PubMed]

Karma A. (1994). Electrical alternans and spiral wave breakup in cardiac tissue. Chaos (Woodbury, N.Y.). 4 [PubMed]

Kim J, Ghosh S, Nunziato DA, Pitt GS. (2004). Identification of the components controlling inactivation of voltage-gated Ca2+ channels. Neuron. 41 [PubMed]

Koller ML, Riccio ML, Gilmour RF. (1998). Dynamic restitution of action potential duration during electrical alternans and ventricular fibrillation. The American journal of physiology. 275 [PubMed]

Liang H et al. (2003). Unified mechanisms of Ca2+ regulation across the Ca2+ channel family. Neuron. 39 [PubMed]

Lo-A-Njoe SM, Wilde AA, van Erven L, Blom NA. (2005). Syndactyly and long QT syndrome (CaV1.2 missense mutation G406R) is associated with hypertrophic cardiomyopathy. Heart rhythm. 2 [PubMed]

Luo CH, Rudy Y. (1991). A model of the ventricular cardiac action potential. Depolarization, repolarization, and their interaction. Circulation research. 68 [PubMed]

Luo CH, Rudy Y. (1994). A dynamic model of the cardiac ventricular action potential. I. Simulations of ionic currents and concentration changes. Circulation research. 74 [PubMed]

Morad M, Soldatov N. (2005). Calcium channel inactivation: possible role in signal transduction and Ca2+ signaling. Cell calcium. 38 [PubMed]

Mori MX, Erickson MG, Yue DT. (2004). Functional stoichiometry and local enrichment of calmodulin interacting with Ca2+ channels. Science (New York, N.Y.). 304 [PubMed]

Nemec J, Ackerman MJ, Tester DJ, Hejlik J, Shen WK. (2003). Catecholamine-provoked microvoltage T wave alternans in genotyped long QT syndrome. Pacing and clinical electrophysiology : PACE. 26 [PubMed]

Nolasco JB, Dahlen RW. (1968). A graphic method for the study of alternation in cardiac action potentials. Journal of applied physiology. 25 [PubMed]

Otani NF, Gilmour RF. (1997). Memory models for the electrical properties of local cardiac systems. Journal of theoretical biology. 187 [PubMed]

Pastore JM, Girouard SD, Laurita KR, Akar FG, Rosenbaum DS. (1999). Mechanism linking T-wave alternans to the genesis of cardiac fibrillation. Circulation. 99 [PubMed]

Pastore JM, Rosenbaum DS. (2000). Role of structural barriers in the mechanism of alternans-induced reentry. Circulation research. 87 [PubMed]

Pitt GS et al. (2001). Molecular basis of calmodulin tethering and Ca2+-dependent inactivation of L-type Ca2+ channels. The Journal of biological chemistry. 276 [PubMed]

Plonsey RC. (1988). Bioelectricity A Quantitative Approach.

Priebe L, Beuckelmann DJ. (1998). Simulation study of cellular electric properties in heart failure. Circulation research. 82 [PubMed]

Qu Z, Garfinkel A, Chen PS, Weiss JN. (2000). Mechanisms of discordant alternans and induction of reentry in simulated cardiac tissue. Circulation. 102 [PubMed]

Satler CA, Vesely MR, Duggal P, Ginsburg GS, Beggs AH. (1998). Multiple different missense mutations in the pore region of HERG in patients with long QT syndrome. Human genetics. 102 [PubMed]

Sato D et al. (2006). Spatially discordant alternans in cardiac tissue: role of calcium cycling. Circulation research. 99 [PubMed]

Sato D et al. (2007). Inferring the cellular origin of voltage and calcium alternans from the spatial scales of phase reversal during discordant alternans. Biophysical journal. 92 [PubMed]

Shi C, Soldatov NM. (2002). Molecular determinants of voltage-dependent slow inactivation of the Ca2+ channel. The Journal of biological chemistry. 277 [PubMed]

Soldatov NM, Oz M, O'Brien KA, Abernethy DR, Morad M. (1998). Molecular determinants of L-type Ca2+ channel inactivation. Segment exchange analysis of the carboxyl-terminal cytoplasmic motif encoded by exons 40-42 of the human alpha1C subunit gene. The Journal of biological chemistry. 273 [PubMed]

Splawski I et al. (2005). Severe arrhythmia disorder caused by cardiac L-type calcium channel mutations. Proceedings of the National Academy of Sciences of the United States of America. 102 [PubMed]

Splawski I et al. (2004). Ca(V)1.2 calcium channel dysfunction causes a multisystem disorder including arrhythmia and autism. Cell. 119 [PubMed]

Stotz SC, Zamponi GW. (2001). Identification of inactivation determinants in the domain IIS6 region of high voltage-activated calcium channels. The Journal of biological chemistry. 276 [PubMed]

Tsien RW, Zuhlke RD, Reuter H, Pitt GS. (2000). Ca2+-sensitive inactivation and facilitation of L-type Ca2+ channels both depend on specific amino acid residues in a consensus calmodulin-binding motif in the(alpha)1C subunit. J Biol Chem. 275

Watanabe MA, Fenton FH, Evans SJ, Hastings HM, Karma A. (2001). Mechanisms for discordant alternans. Journal of cardiovascular electrophysiology. 12 [PubMed]

Zühlke RD, Pitt GS, Deisseroth K, Tsien RW, Reuter H. (1999). Calmodulin supports both inactivation and facilitation of L-type calcium channels. Nature. 399 [PubMed]

ten Tusscher KH, Noble D, Noble PJ, Panfilov AV. (2004). A model for human ventricular tissue. American journal of physiology. Heart and circulatory physiology. 286 [PubMed]

References and models that cite this paper
This website requires cookies and limited processing of your personal data in order to function. By continuing to browse or otherwise use this site, you are agreeing to this use. See our Privacy policy and how to cite and terms of use.