Baker MD. (2005). Protein kinase C mediates up-regulation of tetrodotoxin-resistant, persistent Na+ current in rat and mouse sensory neurones. The Journal of physiology. 567 [PubMed]

• Models of Na channels from a paper on the PKC control of I Na,P (Baker 2005) [Model]

Baker MD, Chandra SY, Ding Y, Waxman SG, Wood JN. (2003). GTP-induced tetrodotoxin-resistant Na+ current regulates excitability in mouse and rat small diameter sensory neurones. The Journal of physiology. 548 [PubMed]

Hennings K, Arendt-Nielsen L, Andersen OK. (2005). Breakdown of accommodation in nerve: a possible role for persistent sodium current. Theoretical biology & medical modelling. 2 [PubMed]

• Breakdown of accmmodation in nerve: a possible role for INAp (Hennings et al 2005) [Model]

Kovalsky Y, Amir R, Devor M. (2008). Subthreshold oscillations facilitate neuropathic spike discharge by overcoming membrane accommodation. Experimental neurology. 210 [PubMed]

Kovalsky Y, Amir R, Devor M. (2009). Simulation in sensory neurons reveals a key role for delayed Na+ current in subthreshold oscillations and ectopic discharge: implications for neuropathic pain. Journal of neurophysiology. 102 [PubMed]

• Dorsal root ganglion (DRG) neuronal model (Kovalsky et al. 2009) [Model]

Kuo JJ, Lee RH, Zhang L, Heckman CJ. (2006). Essential role of the persistent sodium current in spike initiation during slowly rising inputs in mouse spinal neurones. The Journal of physiology. 574 [PubMed]