ModelDB: Paper information

Wallace RH et al. (2001). Mutant GABA(A) receptor gamma2-subunit in childhood absence epilepsy and febrile seizures. Nature genetics. 28 [PubMed]

See more from authors: Wallace RH · Marini C · Petrou S · Harkin LA · Bowser DN · Panchal RG · Williams DA · Sutherland GR · Mulley JC · Scheffer IE · Berkovic SF

References and models cited by this paper

References and models that cite this paper

Barela AJ et al. (2006). An epilepsy mutation in the sodium channel SCN1A that decreases channel excitability. The Journal of neuroscience : the official journal of the Society for Neuroscience. 26 [PubMed]

Sodium channel mutations causing generalized epilepsy with febrile seizures + (Barela et al. 2006) [Model]

Hall SP et al. (2018). Enhanced interlaminar excitation or reduced superficial layer inhibition in neocortex generates different spike-and-wave-like electrographic events in vitro. Journal of neurophysiology. 119 [PubMed]

The origin of different spike and wave-like events (Hall et al 2017) [Model]

Lema GM, Auerbach A. (2006). Modes and models of GABA(A) receptor gating. The Journal of physiology. 572 [PubMed]

Liu Y, Milton J, Campbell SA. (2019). Outgrowing seizures in Childhood Absence Epilepsy: time delays and bistability. Journal of computational neuroscience. 46 [PubMed]

Thalamocortical loop with delay for investigation of absence epilepsy (Liu et al 2019) [Model]

Thomas EA, Reid CA, Petrou S. (2010). Mossy fiber sprouting interacts with sodium channel mutations to increase dentate gyrus excitability. Epilepsia. 51 [PubMed]

Na channel mutations in the dentate gyrus (Thomas et al. 2009) [Model]