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• Alcohol excites Cerebellar Golgi Cells by inhibiting the Na+/K+ ATPase (Botta et al.2010) [Model]

Cayco-Gajic NA, Clopath C, Silver RA. (2017). Sparse synaptic connectivity is required for decorrelation and pattern separation in feedforward networks. Nature communications. 8 [PubMed]

• Sparse connectivity is required for decorrelation, pattern separation (Cayco-Gajic et al 2017) [Model]

De Schutter E, Simoes-de-Souza FM. (2011). Robustness effect of gap junctions between Golgi cells on cerebellar cortex oscillations Neural Systems & Circuits. 1:7

• Cerebellar cortex oscil. robustness from Golgi cell gap jncs (Simoes de Souza and De Schutter 2011) [Model]

Diwakar S, Lombardo P, Solinas S, Naldi G, D'Angelo E. (2011). Local field potential modeling predicts dense activation in cerebellar granule cells clusters under LTP and LTD control. PloS one. 6 [PubMed]

• Reconstructing cerebellar granule layer evoked LFP using convolution (ReConv) (Diwakar et al. 2011) [Model]

Forrest MD. (2015). Simulation of alcohol action upon a detailed Purkinje neuron model and a simpler surrogate model that runs >400 times faster. BMC neuroscience. 16 [PubMed]

• Alcohol action in a detailed Purkinje neuron model and an efficient simplified model (Forrest 2015) [Model]

Garrido JA, Ros E, D'Angelo E. (2013). Spike timing regulation on the millisecond scale by distributed synaptic plasticity at the cerebellum input stage: a simulation study. Frontiers in computational neuroscience. 7 [PubMed]

• Distributed synaptic plasticity and spike timing (Garrido et al. 2013) [Model]

Geminiani A, Casellato C, Antonietti A, D'Angelo E, Pedrocchi A. (2018). A Multiple-Plasticity Spiking Neural Network Embedded in a Closed-Loop Control System to Model Cerebellar Pathologies. International journal of neural systems. 28 [PubMed]

Khatri SN, Wu WC, Yang Y, Pugh JR. (2019). Mechanisms of GABAB receptor enhancement of extrasynaptic GABAA receptor currents in cerebellar granule cells. Scientific reports. 9 [PubMed]

Ovsepian SV et al. (2013). A defined heteromeric KV1 channel stabilizes the intrinsic pacemaking and regulates the output of deep cerebellar nuclear neurons to thalamic targets. The Journal of physiology. 591 [PubMed]

• KV1 channel governs cerebellar output to thalamus (Ovsepian et al. 2013) [Model]