Chen M et al. (2015). Critical Roles of the Direct GABAergic Pallido-cortical Pathway in Controlling Absence Seizures. PLoS computational biology. 11 [PubMed]

• Basal ganglia-corticothalamic (BGCT) network (Chen et al., 2014) [Model]

Chen M et al. (2014). Bidirectional control of absence seizures by the basal ganglia: a computational evidence. PLoS computational biology. 10 [PubMed]

• Basal ganglia-corticothalamic (BGCT) network (Chen et al., 2014) [Model]

Grado LL, Johnson MD, Netoff TI. (2018). Bayesian adaptive dual control of deep brain stimulation in a computational model of Parkinson's disease. PLoS computational biology. 14 [PubMed]

• Adaptive dual control of deep brain stimulation in Parkinsons disease simulations (Grado et al 2018) [Model]

Hu B et al. (2017). Control of absence epilepsy seizures in specific relay nuclei of thalamus. Journal of theoretical biology. 435 [PubMed]

Kerr CC et al. (2013). Cortical information flow in Parkinson's disease: a composite network/field model. Frontiers in computational neuroscience. 7 [PubMed]

• Composite spiking network/neural field model of Parkinsons (Kerr et al 2013) [Model]

Mulcahy G, Atwood B, Kuznetsov A. (2020). Basal ganglia role in learning rewarded actions and executing previously learned choices: Healthy and diseased states. PloS one. 15 [PubMed]

• Cortico - Basal Ganglia Loop (Mulcahy et al 2020) [Model]

Pavlides A, Hogan SJ, Bogacz R. (2015). Computational Models Describing Possible Mechanisms for Generation of Excessive Beta Oscillations in Parkinson's Disease. PLoS computational biology. 11 [PubMed]

• Excessive beta oscillations in Parkinson's disease (Pavlides et al. 2015) [Model]