Balbi P, Massobrio P, Hellgren Kotaleski J. (2017). A single Markov-type kinetic model accounting for the macroscopic currents of all human voltage-gated sodium channel isoforms. PLoS computational biology. 13 [PubMed]
Behn CG, Brown EN, Scammell TE, Kopell NJ. (2007). Mathematical model of network dynamics governing mouse sleep-wake behavior. Journal of neurophysiology. 97 [PubMed]
Boucher PA, Joós B, Morris CE. (2012). Coupled left-shift of Nav channels: modeling the Na?-loading and dysfunctional excitability of damaged axons. Journal of computational neuroscience. 33 [PubMed]
Chan CS, Shigemoto R, Mercer JN, Surmeier DJ. (2004). HCN2 and HCN1 channels govern the regularity of autonomous pacemaking and synaptic resetting in globus pallidus neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 24 [PubMed]
Doiron B, Noonan L, Lemon N, Turner RW. (2003). Persistent Na+ current modifies burst discharge by regulating conditional backpropagation of dendritic spikes. Journal of neurophysiology. 89 [PubMed]
Fleidervish IA, Libman L, Katz E, Gutnick MJ. (2008). Endogenous polyamines regulate cortical neuronal excitability by blocking voltage-gated Na+ channels. Proceedings of the National Academy of Sciences of the United States of America. 105 [PubMed]
Hsu CL, Zhao X, Milstein AD, Spruston N. (2018). Persistent sodium current mediates the steep voltage dependence of spatial coding in hippocampal pyramidal neurons Neuron. 99
Häusser M, Monsivais P. (2003). Less means more: inhibition of spontaneous firing triggers persistent increases in excitability. Neuron. 40 [PubMed]
Häusser M et al. (2004). The beat goes on: spontaneous firing in mammalian neuronal microcircuits. The Journal of neuroscience : the official journal of the Society for Neuroscience. 24 [PubMed]
Jaffe DB, Brenner R. (2018). A computational model for how the fast afterhyperpolarization paradoxically increases gain in regularly firing neurons. Journal of neurophysiology. 119 [PubMed]
Kole MH et al. (2008). Action potential generation requires a high sodium channel density in the axon initial segment. Nature neuroscience. 11 [PubMed]
Kuo JJ, Lee RH, Zhang L, Heckman CJ. (2006). Essential role of the persistent sodium current in spike initiation during slowly rising inputs in mouse spinal neurones. The Journal of physiology. 574 [PubMed]
Maurice N et al. (2004). D2 dopamine receptor-mediated modulation of voltage-dependent Na+ channels reduces autonomous activity in striatal cholinergic interneurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 24 [PubMed]
Mercer JN, Chan CS, Tkatch T, Held J, Surmeier DJ. (2007). Nav1.6 sodium channels are critical to pacemaking and fast spiking in globus pallidus neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 27 [PubMed]
Spratt PWE et al. (2021). Paradoxical hyperexcitability from NaV1.2 sodium channel loss in neocortical pyramidal cells Cell reports. 36 [PubMed]
Venugopal S et al. (2019). Resurgent Na+ Current Offers Noise Modulation in Bursting Neurons. PLoS computational biology. 15 [PubMed]
Williams SR, Stuart GJ. (2003). Voltage- and site-dependent control of the somatic impact of dendritic IPSPs. The Journal of neuroscience : the official journal of the Society for Neuroscience. 23 [PubMed]