ModelDB: Paper information

Soldovieri MV et al. (2007). Atypical gating of M-type potassium channels conferred by mutations in uncharged residues in the S4 region of KCNQ2 causing benign familial neonatal convulsions. The Journal of neuroscience : the official journal of the Society for Neuroscience. 27 [PubMed]

See more from authors: Soldovieri MV · Cilio MR · Miceli F · Bellini G · Miraglia del Giudice E · Castaldo P · Hernandez CC · Shapiro MS · Pascotto A · Annunziato L · Taglialatela M

References and models cited by this paper

References and models that cite this paper

Ferrante M, Blackwell KT, Migliore M, Ascoli GA. (2008). Computational models of neuronal biophysics and the characterization of potential neuropharmacological targets. Current medicinal chemistry. 15 [PubMed]

Computational neuropharmacology of CA1 pyramidal neuron (Ferrante et al. 2008) [Model]

Miceli F et al. (2015). Early-onset epileptic encephalopathy caused by gain-of-function mutations in the voltage sensor of Kv7.2 and Kv7.3 potassium channel subunits. The Journal of neuroscience : the official journal of the Society for Neuroscience. 35 [PubMed]

Early-onset epileptic encephalopathy (Miceli et al. 2015) [Model]

Miceli F et al. (2009). Neutralization of a unique, negatively-charged residue in the voltage sensor of K V 7.2 subunits in a sporadic case of benign familial neonatal seizures. Neurobiology of disease. 34 [PubMed]

CA1 pyramidal neurons: effects of a Kv7.2 mutation (Miceli et al. 2009) [Model]