Spine neck plasticity controls postsynaptic calcium signals (Grunditz et al. 2008)

This model was set up to dissect the relative contribution of different channels to the spine calcium transients measured at single spines.

Model Type: Synapse

Cell Type(s): Hippocampus CA1 pyramidal GLU cell

Currents: I Calcium; I R

Receptors: AMPA; NMDA

Transmitters: Glutamate

Model Concept(s): Calcium dynamics

Simulation Environment: NEURON

Implementer(s): Mueller, Asa [asa.mueller at fmi.ch]

References:

Grunditz A, Holbro N, Tian L, Zuo Y, Oertner TG. (2008). Spine neck plasticity controls postsynaptic calcium signals through electrical compartmentalization. The Journal of neuroscience : the official journal of the Society for Neuroscience. 28 [PubMed]