Aizawa Y et al. (2007). A novel mutation in KCNQ1 associated with a potent dominant negative effect as the basis for the LQT1 form of the long QT syndrome. Journal of cardiovascular electrophysiology. 18 [PubMed]
Bosch RF et al. (1998). Effects of the chromanol 293B, a selective blocker of the slow, component of the delayed rectifier K+ current, on repolarization in human and guinea pig ventricular myocytes. Cardiovascular research. 38 [PubMed]
Brette F, Le Guennec JY, Leroy J, Salle L. (2007). Ca2+ currents in cardiac myocytes: Old story, new insights. Prog Biophys Mol Biol. 91
Carter S, Colyer J, Sitsapesan R. (2006). Maximum phosphorylation of the cardiac ryanodine receptor at serine-2809 by protein kinase a produces unique modifications to channel gating and conductance not observed at lower levels of phosphorylation. Circulation research. 98 [PubMed]
Chen H, Kim LA, Rajan S, Xu S, Goldstein SA. (2003). Charybdotoxin binding in the I(Ks) pore demonstrates two MinK subunits in each channel complex. Neuron. 40 [PubMed]
Chouabe C, Drici MD, Romey G, Barhanin J, Lazdunski M. (1998). HERG and KvLQT1/IsK, the cardiac K+ channels involved in long QT syndromes, are targets for calcium channel blockers. Molecular pharmacology. 54 [PubMed]
Derkach V, Barria A, Soderling TR. (1999). Ca2+/calmodulin-kinase II enhances channel conductance of alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionate type glutamate receptors. Proceedings of the National Academy of Sciences of the United States of America. 96 [PubMed]
Dilly KW et al. (2004). Overexpression of beta2-adrenergic receptors cAMP-dependent protein kinase phosphorylates and modulates slow delayed rectifier potassium channels expressed in murine heart: evidence for receptor/channel co-localization. The Journal of biological chemistry. 279 [PubMed]
Faber GM, Rudy Y. (2000). Action potential and contractility changes in [Na(+)](i) overloaded cardiac myocytes: a simulation study. Biophysical journal. 78 [PubMed]
Imredy JP, Penniman JR, Dech SJ, Irving WD, Salata JJ. (2008). Modeling of the adrenergic response of the human IKs current (hKCNQ1/hKCNE1) stably expressed in HEK-293 cells. American journal of physiology. Heart and circulatory physiology. 295 [PubMed]
Iost N et al. (1998). Delayed rectifier potassium current in undiseased human ventricular myocytes. Cardiovascular research. 40 [PubMed]
Lu Z, Kamiya K, Opthof T, Yasui K, Kodama I. (2001). Density and kinetics of I(Kr) and I(Ks) in guinea pig and rabbit ventricular myocytes explain different efficacy of I(Ks) blockade at high heart rate in guinea pig and rabbit: implications for arrhythmogenesis in humans. Circulation. 104 [PubMed]
Marx SO et al. (2002). Requirement of a macromolecular signaling complex for beta adrenergic receptor modulation of the KCNQ1-KCNE1 potassium channel. Science (New York, N.Y.). 295 [PubMed]
Moreno AP, Sáez JC, Fishman GI, Spray DC. (1994). Human connexin43 gap junction channels. Regulation of unitary conductances by phosphorylation. Circulation research. 74 [PubMed]
Morin TJ, Kobertz WR. (2008). Counting membrane-embedded KCNE beta-subunits in functioning K+ channel complexes. Proceedings of the National Academy of Sciences of the United States of America. 105 [PubMed]
Rocchetti M, Besana A, Gurrola GB, Possani LD, Zaza A. (2001). Rate dependency of delayed rectifier currents during the guinea-pig ventricular action potential. The Journal of physiology. 534 [PubMed]
Rocchetti M et al. (2006). Rate dependency of beta-adrenergic modulation of repolarizing currents in the guinea-pig ventricle. The Journal of physiology. 574 [PubMed]
Sanguinetti MC, Jurkiewicz NK, Scott A, Siegl PK. (1991). Isoproterenol antagonizes prolongation of refractory period by the class III antiarrhythmic agent E-4031 in guinea pig myocytes. Mechanism of action. Circulation research. 68 [PubMed]
Saucerman JJ, Healy SN, Belik ME, Puglisi JL, McCulloch AD. (2004). Proarrhythmic consequences of a KCNQ1 AKAP-binding domain mutation: computational models of whole cells and heterogeneous tissue. Circulation research. 95 [PubMed]
Schwartz PJ et al. (2001). Genotype-phenotype correlation in the long-QT syndrome: gene-specific triggers for life-threatening arrhythmias. Circulation. 103 [PubMed]
Seebohm G et al. (2007). Regulation of endocytic recycling of KCNQ1/KCNE1 potassium channels. Circulation research. 100 [PubMed]
Silva J, Rudy Y. (2005). Subunit interaction determines IKs participation in cardiac repolarization and repolarization reserve. Circulation. 112 [PubMed]
Silverman WR, Roux B, Papazian DM. (2003). Structural basis of two-stage voltage-dependent activation in K+ channels. Proceedings of the National Academy of Sciences of the United States of America. 100 [PubMed]
Terrenoire C, Clancy CE, Cormier JW, Sampson KJ, Kass RS. (2005). Autonomic control of cardiac action potentials: role of potassium channel kinetics in response to sympathetic stimulation. Circulation research. 96 [PubMed]
Tzounopoulos T, Maylie J, Adelman JP. (1998). Gating of I(sK) channels expressed in Xenopus oocytes. Biophysical journal. 74 [PubMed]
Varro A et al. (2000). The role of the delayed rectifier component IKs in dog ventricular muscle and Purkinje fibre repolarization. The Journal of physiology. 523 Pt 1 [PubMed]
Volders PG et al. (2003). Probing the contribution of IKs to canine ventricular repolarization: key role for beta-adrenergic receptor stimulation. Circulation. 107 [PubMed]
Walsh KB, Kass RS. (1991). Distinct voltage-dependent regulation of a heart-delayed IK by protein kinases A and C. The American journal of physiology. 261 [PubMed]
Wang Q et al. (1996). Positional cloning of a novel potassium channel gene: KVLQT1 mutations cause cardiac arrhythmias. Nature genetics. 12 [PubMed]