Graupner M, Brunel N. (2007). STDP in a bistable synapse model based on CaMKII and associated signaling pathways. PLoS computational biology. 3 [PubMed]

• CaMKII system exhibiting bistability with respect to calcium (Graupner and Brunel 2007) [Model]

Graupner M, Brunel N. (2012). Calcium-based plasticity model explains sensitivity of synaptic changes to spike pattern, rate, and dendritic location. Proceedings of the National Academy of Sciences of the United States of America. 109 [PubMed]

Gütig R, Sompolinsky H. (2009). Time-warp-invariant neuronal processing. PLoS biology. 7 [PubMed]

• Time-warp-invariant neuronal processing (Gutig & Sompolinsky 2009) [Model]

Kim Y, Hsu CL, Cembrowski MS, Mensh BD, Spruston N. (2015). Dendritic sodium spikes are required for long-term potentiation at distal synapses on hippocampal pyramidal neurons. eLife. 4 [PubMed]

• CA1 pyramidal neuron: Dendritic Na+ spikes are required for LTP at distal synapses (Kim et al 2015) [Model]

Saudargiene A, Cobb S, Graham BP. (2015). A computational study on plasticity during theta cycles at Schaffer collateral synapses on CA1 pyramidal cells in the hippocampus. Hippocampus. 25 [PubMed]

• CA1 pyramidal neuron: synaptic plasticity during theta cycles (Saudargiene et al. 2015) [Model]

Urakubo H, Honda M, Froemke RC, Kuroda S. (2008). Requirement of an allosteric kinetics of NMDA receptors for spike timing-dependent plasticity. The Journal of neuroscience : the official journal of the Society for Neuroscience. 28 [PubMed]

• An allosteric kinetics of NMDARs in STDP (Urakubo et al. 2008) [Model]

Yu X, Shouval HZ, Knierim JJ. (2008). A biophysical model of synaptic plasticity and metaplasticity can account for the dynamics of the backward shift of hippocampal place fields. Journal of neurophysiology. 100 [PubMed]