ModelDB: Paper information

Schwartz PJ et al. (1995). Long QT syndrome patients with mutations of the SCN5A and HERG genes have differential responses to Na+ channel blockade and to increases in heart rate. Implications for gene-specific therapy. Circulation. 92 [PubMed]

See more from authors: Schwartz PJ · Priori SG · Locati EH · Napolitano C · Cantù F · Towbin JA · Keating MT · Hammoude H · Brown AM · Chen LS · Colatsky TJ

References and models cited by this paper

References and models that cite this paper

Clancy CE, Rudy Y. (1999). Linking a genetic defect to its cellular phenotype in a cardiac arrhythmia. Nature. 400 [PubMed]

Ventricular cell model (Guinea-pig-type) (Luo, Rudy 1991, +11 other papers!) (C++) [Model]

Clancy CE, Rudy Y. (2001). Cellular consequences of HERG mutations in the long QT syndrome: precursors to sudden cardiac death. Cardiovascular research. 50 [PubMed]

Consequences of HERG mutations in the long QT syndrome (Clancy, Rudy 2001) [Model]

Mazhari R, Greenstein JL, Winslow RL, Marbán E, Nuss HB. (2001). Molecular interactions between two long-QT syndrome gene products, HERG and KCNE2, rationalized by in vitro and in silico analysis. Circulation research. 89 [PubMed]