Barela AJ et al. (2006). An epilepsy mutation in the sodium channel SCN1A that decreases channel excitability. The Journal of neuroscience : the official journal of the Society for Neuroscience. 26 [PubMed]

• Sodium channel mutations causing generalized epilepsy with febrile seizures + (Barela et al. 2006) [Model]

Clancy CE, Kass RS. (2004). Theoretical investigation of the neuronal Na+ channel SCN1A: abnormal gating and epilepsy. Biophysical journal. 86 [PubMed]

• Markov models of SCN1A (NaV1.1) applied to abnormal gating and epilepsy (Clancy and Kass 2004) [Model]

Kahlig KM, Misra SN, George AL. (2006). Impaired inactivation gate stabilization predicts increased persistent current for an epilepsy-associated SCN1A mutation. The Journal of neuroscience : the official journal of the Society for Neuroscience. 26 [PubMed]

Kovalsky Y, Amir R, Devor M. (2009). Simulation in sensory neurons reveals a key role for delayed Na+ current in subthreshold oscillations and ectopic discharge: implications for neuropathic pain. Journal of neurophysiology. 102 [PubMed]

• Dorsal root ganglion (DRG) neuronal model (Kovalsky et al. 2009) [Model]

Vitko I et al. (2005). Functional characterization and neuronal modeling of the effects of childhood absence epilepsy variants of CACNA1H, a T-type calcium channel. The Journal of neuroscience : the official journal of the Society for Neuroscience. 25 [PubMed]

• T channel currents (Vitko et al 2005) [Model]