Broicher T et al. (2007). Specific expression of low-voltage-activated calcium channel isoforms and splice variants in thalamic local circuit interneurons. Molecular and cellular neurosciences. 36 [PubMed]

Broicher T, Kanyshkova T, Meuth P, Pape HC, Budde T. (2008). Correlation of T-channel coding gene expression, IT, and the low threshold Ca2+ spike in the thalamus of a rat model of absence epilepsy. Molecular and cellular neurosciences. 39 [PubMed]

Broicher T et al. (2007). T-current related effects of antiepileptic drugs and a Ca2+ channel antagonist on thalamic relay and local circuit interneurons in a rat model of absence epilepsy. Neuropharmacology. 53 [PubMed]

Chen M et al. (2015). Critical Roles of the Direct GABAergic Pallido-cortical Pathway in Controlling Absence Seizures. PLoS computational biology. 11 [PubMed]

• Basal ganglia-corticothalamic (BGCT) network (Chen et al., 2014) [Model]

Chen M et al. (2014). Bidirectional control of absence seizures by the basal ganglia: a computational evidence. PLoS computational biology. 10 [PubMed]

• Basal ganglia-corticothalamic (BGCT) network (Chen et al., 2014) [Model]

Hall S et al. (2015). Unbalanced Peptidergic Inhibition in Superficial Neocortex Underlies Spike and Wave Seizure Activity. The Journal of neuroscience : the official journal of the Society for Neuroscience. 35 [PubMed]

• Unbalanced peptidergic inhibition in superficial cortex underlies seizure activity (Hall et al 2015) [Model]
• Electrodecrements in in vitro model of infantile spasms (Traub et al 2020) [Model]

Hall SP et al. (2018). Enhanced interlaminar excitation or reduced superficial layer inhibition in neocortex generates different spike-and-wave-like electrographic events in vitro. Journal of neurophysiology. 119 [PubMed]

• The origin of different spike and wave-like events (Hall et al 2017) [Model]

Hayut I, Fanselow EE, Connors BW, Golomb D. (2011). LTS and FS inhibitory interneurons, short-term synaptic plasticity, and cortical circuit dynamics. PLoS computational biology. 7 [PubMed]

• Rate model of a cortical RS-FS-LTS network (Hayut et al. 2011) [Model]

Liu Y, Milton J, Campbell SA. (2019). Outgrowing seizures in Childhood Absence Epilepsy: time delays and bistability. Journal of computational neuroscience. 46 [PubMed]

• Thalamocortical loop with delay for investigation of absence epilepsy (Liu et al 2019) [Model]

Suffczynski P, Kalitzin S, Lopes Da Silva FH. (2004). Dynamics of non-convulsive epileptic phenomena modeled by a bistable neuronal network. Neuroscience. 126 [PubMed]

• Thalamocortical model of spike and wave seizures (Suffczynski et al. 2004) [Model]

Vitko I et al. (2005). Functional characterization and neuronal modeling of the effects of childhood absence epilepsy variants of CACNA1H, a T-type calcium channel. The Journal of neuroscience : the official journal of the Society for Neuroscience. 25 [PubMed]

• T channel currents (Vitko et al 2005) [Model]

Zhao X, Robinson PA. (2015). Generalized seizures in a neural field model with bursting dynamics. Journal of computational neuroscience. 39 [PubMed]

• Neural-field model of generalized seizures (Zhao et al., 2015) [Model]