Meuth SG et al. (2008). TWIK-related acid-sensitive K+ channel 1 (TASK1) and TASK3 critically influence T lymphocyte effector functions. The Journal of biological chemistry. 283 [PubMed]

See more from authors: Meuth SG · Bittner S · Meuth P · Simon OJ · Budde T · Wiendl H

References and models cited by this paper

Bean BP. (2007). The action potential in mammalian central neurons. Nature reviews. Neuroscience. 8 [PubMed]

Beeton C et al. (2001). Selective blocking of voltage-gated K+ channels improves experimental autoimmune encephalomyelitis and inhibits T cell activation. Journal of immunology (Baltimore, Md. : 1950). 166 [PubMed]

Beeton C, Chandy KG. (2005). Potassium channels, memory T cells, and multiple sclerosis. The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry. 11 [PubMed]

Beeton C et al. (2005). Targeting effector memory T cells with a selective peptide inhibitor of Kv1.3 channels for therapy of autoimmune diseases. Molecular pharmacology. 67 [PubMed]

Beeton C et al. (2001). Selective blockade of T lymphocyte K(+) channels ameliorates experimental autoimmune encephalomyelitis, a model for multiple sclerosis. Proceedings of the National Academy of Sciences of the United States of America. 98 [PubMed]

Beeton C et al. (2006). Kv1.3 channels are a therapeutic target for T cell-mediated autoimmune diseases. Proceedings of the National Academy of Sciences of the United States of America. 103 [PubMed]

Berg AP, Bayliss DA. (2007). Striatal cholinergic interneurons express a receptor-insensitive homomeric TASK-3-like background K+ current. Journal of neurophysiology. 97 [PubMed]

Brown DA. (2000). Neurobiology: the acid test for resting potassium channels. Current biology : CB. 10 [PubMed]

Buckler KJ, Williams BA, Honore E. (2000). An oxygen-, acid- and anaesthetic-sensitive TASK-like background potassium channel in rat arterial chemoreceptor cells. The Journal of physiology. 525 Pt 1 [PubMed]

Cahalan MD, Chandy KG. (1997). Ion channels in the immune system as targets for immunosuppression. Current opinion in biotechnology. 8 [PubMed]

Cahalan MD, Wulff H, Chandy KG. (2001). Molecular properties and physiological roles of ion channels in the immune system. Journal of clinical immunology. 21 [PubMed]

Chandy KG et al. (2001). Potassium channels in T lymphocytes: toxins to therapeutic immunosuppressants. Toxicon : official journal of the International Society on Toxinology. 39 [PubMed]

Chandy KG, DeCoursey TE, Cahalan MD, McLaughlin C, Gupta S. (1984). Voltage-gated potassium channels are required for human T lymphocyte activation. The Journal of experimental medicine. 160 [PubMed]

Chandy KG et al. (2004). K+ channels as targets for specific immunomodulation. Trends in pharmacological sciences. 25 [PubMed]

Chen WC, Davis RL. (2006). Voltage-gated and two-pore-domain potassium channels in murine spiral ganglion neurons. Hearing research. 222 [PubMed]

Chen X et al. (2006). Inhibition of a background potassium channel by Gq protein alpha-subunits. Proceedings of the National Academy of Sciences of the United States of America. 103 [PubMed]

Czirják G, Enyedi P. (2003). Ruthenium red inhibits TASK-3 potassium channel by interconnecting glutamate 70 of the two subunits. Molecular pharmacology. 63 [PubMed]

De Schutter E, Bower JM. (1994). An active membrane model of the cerebellar Purkinje cell. I. Simulation of current clamps in slice. Journal of neurophysiology. 71 [PubMed]

DeCoursey TE, Chandy KG, Gupta S, Cahalan MD. (1984). Voltage-gated K+ channels in human T lymphocytes: a role in mitogenesis? Nature. 307 [PubMed]

Dodt HU, Zieglgänsberger W. (1990). Visualizing unstained neurons in living brain slices by infrared DIC-videomicroscopy. Brain research. 537 [PubMed]

Ghanshani S et al. (2000). Up-regulation of the IKCa1 potassium channel during T-cell activation. Molecular mechanism and functional consequences. The Journal of biological chemistry. 275 [PubMed]

Goldstein SA, Bockenhauer D, O'Kelly I, Zilberberg N. (2001). Potassium leak channels and the KCNK family of two-P-domain subunits. Nature reviews. Neuroscience. 2 [PubMed]

Grissmer S, Nguyen AN, Cahalan MD. (1993). Calcium-activated potassium channels in resting and activated human T lymphocytes. Expression levels, calcium dependence, ion selectivity, and pharmacology. The Journal of general physiology. 102 [PubMed]

Hines ML, Carnevale NT. (1997). The NEURON simulation environment. Neural computation. 9 [PubMed]

Kang D, Choe C, Kim D. (2004). Functional expression of TREK-2 in insulin-secreting MIN6 cells. Biochemical and biophysical research communications. 323 [PubMed]

Kleinschnitz C, Meuth SG, Kieseier BC, Wiendl H. (2007). Immunotherapeutic approaches in MS: update on pathophysiology and emerging agents or strategies 2006. Endocrine, metabolic & immune disorders drug targets. 7 [PubMed]

Leonoudakis D et al. (1998). An open rectifier potassium channel with two pore domains in tandem cloned from rat cerebellum. The Journal of neuroscience : the official journal of the Society for Neuroscience. 18 [PubMed]

Lesage F. (2003). Pharmacology of neuronal background potassium channels. Neuropharmacology. 44 [PubMed]

Lewis RS, Cahalan MD. (1995). Potassium and calcium channels in lymphocytes. Annual review of immunology. 13 [PubMed]

Logsdon NJ, Kang J, Togo JA, Christian EP, Aiyar J. (1997). A novel gene, hKCa4, encodes the calcium-activated potassium channel in human T lymphocytes. The Journal of biological chemistry. 272 [PubMed]

Maingret F, Patel AJ, Lazdunski M, Honoré E. (2001). The endocannabinoid anandamide is a direct and selective blocker of the background K(+) channel TASK-1. The EMBO journal. 20 [PubMed]

Mathie A. (2007). Neuronal two-pore-domain potassium channels and their regulation by G protein-coupled receptors. The Journal of physiology. 578 [PubMed]

Meadows HJ, Randall AD. (2001). Functional characterisation of human TASK-3, an acid-sensitive two-pore domain potassium channel. Neuropharmacology. 40 [PubMed]

Meuth SG et al. (2006). The contribution of TWIK-related acid-sensitive K+-containing channels to the function of dorsal lateral geniculate thalamocortical relay neurons. Molecular pharmacology. 69 [PubMed]

Meuth SG et al. (2003). Contribution of TWIK-related acid-sensitive K+ channel 1 (TASK1) and TASK3 channels to the control of activity modes in thalamocortical neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 23 [PubMed]

Meuth SG et al. (2006). Membrane resting potential of thalamocortical relay neurons is shaped by the interaction among TASK3 and HCN2 channels. Journal of neurophysiology. 96 [PubMed]

Meuth SG et al. (2008). CNS inflammation and neuronal degeneration is aggravated by impaired CD200-CD200R-mediated macrophage silencing. Journal of neuroimmunology. 194 [PubMed]

Musset B et al. (2006). Effects of divalent cations and spermine on the K+ channel TASK-3 and on the outward current in thalamic neurons. The Journal of physiology. 572 [PubMed]

Negulescu PA, Shastri N, Cahalan MD. (1994). Intracellular calcium dependence of gene expression in single T lymphocytes. Proceedings of the National Academy of Sciences of the United States of America. 91 [PubMed]

Neher E. (1992). Correction for liquid junction potentials in patch clamp experiments. Methods in enzymology. 207 [PubMed]

Panyi G, Vámosi G, Bodnár A, Gáspár R, Damjanovich S. (2004). Looking through ion channels: recharged concepts in T-cell signaling. Trends in immunology. 25 [PubMed]

Prakriya M, Lewis RS. (2003). CRAC channels: activation, permeation, and the search for a molecular identity. Cell calcium. 33 [PubMed]

Prakriya M, Lewis RS. (2006). Regulation of CRAC channel activity by recruitment of silent channels to a high open-probability gating mode. The Journal of general physiology. 128 [PubMed]

Rajan S et al. (2002). Interaction with 14-3-3 proteins promotes functional expression of the potassium channels TASK-1 and TASK-3. The Journal of physiology. 545 [PubMed]

Sabath DE, Monos DS, Lee SC, Deutsch C, Prystowsky MB. (1986). Cloned T-cell proliferation and synthesis of specific proteins are inhibited by quinine. Proceedings of the National Academy of Sciences of the United States of America. 83 [PubMed]

Schell SR, Nelson DJ, Fozzard HA, Fitch FW. (1987). The inhibitory effects of K+ channel-blocking agents on T lymphocyte proliferation and lymphokine production are "nonspecific". Journal of immunology (Baltimore, Md. : 1950). 139 [PubMed]

Schmidt J et al. (2003). Drug targeting by long-circulating liposomal glucocorticosteroids increases therapeutic efficacy in a model of multiple sclerosis. Brain : a journal of neurology. 126 [PubMed]

Schwarz EC et al. (2007). Calcium dependence of T cell proliferation following focal stimulation. European journal of immunology. 37 [PubMed]

Teisseyre A, Mozrzymas JW. (2006). Influence of extracellular pH on the modulatory effect of zinc ions on Kv1.3 potassium channels. Journal of physiology and pharmacology : an official journal of the Polish Physiological Society. 57 [PubMed]

Teisseyre A, Mozrzymas JW. (2007). The influence of protons and zinc ions on the steady-state inactivation of Kv1.3 potassium channels. Cellular & molecular biology letters. 12 [PubMed]

Tischner D et al. (2006). Polyclonal expansion of regulatory T cells interferes with effector cell migration in a model of multiple sclerosis. Brain : a journal of neurology. 129 [PubMed]

Torborg CL, Berg AP, Jeffries BW, Bayliss DA, McBain CJ. (2006). TASK-like conductances are present within hippocampal CA1 stratum oriens interneuron subpopulations. The Journal of neuroscience : the official journal of the Society for Neuroscience. 26 [PubMed]

Vennekamp J et al. (2004). Kv1.3-blocking 5-phenylalkoxypsoralens: a new class of immunomodulators. Molecular pharmacology. 65 [PubMed]

Wulff H, Beeton C, Chandy KG. (2003). Potassium channels as therapeutic targets for autoimmune disorders. Current opinion in drug discovery & development. 6 [PubMed]

Wulff H et al. (2003). The voltage-gated Kv1.3 K(+) channel in effector memory T cells as new target for MS. The Journal of clinical investigation. 111 [PubMed]

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