Bentwich J et al. (2011). Beneficial effect of repetitive transcranial magnetic stimulation combined with cognitive training for the treatment of Alzheimer's disease: a proof of concept study. Journal of neural transmission (Vienna, Austria : 1996). 118 [PubMed]
Billings LM, Green KN, McGaugh JL, LaFerla FM. (2007). Learning decreases A beta*56 and tau pathology and ameliorates behavioral decline in 3xTg-AD mice. The Journal of neuroscience : the official journal of the Society for Neuroscience. 27 [PubMed]
Csicsvari J, Hirase H, Czurkó A, Mamiya A, Buzsáki G. (1999). Oscillatory coupling of hippocampal pyramidal cells and interneurons in the behaving Rat. The Journal of neuroscience : the official journal of the Society for Neuroscience. 19 [PubMed]
Eckert A et al. (2008). Soluble beta-amyloid leads to mitochondrial defects in amyloid precursor protein and tau transgenic mice. Neuro-degenerative diseases. 5 [PubMed]
Ferrante M, Blackwell KT, Migliore M, Ascoli GA. (2008). Computational models of neuronal biophysics and the characterization of potential neuropharmacological targets. Current medicinal chemistry. 15 [PubMed]
Gasparini S, Migliore M, Magee JC. (2004). On the initiation and propagation of dendritic spikes in CA1 pyramidal neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 24 [PubMed]
Goedert M. (1987). Neuronal localization of amyloid beta protein precursor mRNA in normal human brain and in Alzheimer's disease. The EMBO journal. 6 [PubMed]
Good TA, Smith DO, Murphy RM. (1996). Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons. Biophysical journal. 70 [PubMed]
Hamos JE, DeGennaro LJ, Drachman DA. (1989). Synaptic loss in Alzheimer's disease and other dementias. Neurology. 39 [PubMed]
Hasselmo ME. (1997). A computational model of the progression of Alzheimer's disease. M.D. computing : computers in medical practice. 14 [PubMed]
Hines ML, Carnevale NT. (1997). The NEURON simulation environment. Neural computation. 9 [PubMed]
Horn D, Levy N, Ruppin E. (1996). Neuronal-based synaptic compensation: a computational study in Alzheimer's disease. Neural computation. 8 [PubMed]
Iwatsubo T et al. (1994). Visualization of A beta 42(43) and A beta 40 in senile plaques with end-specific A beta monoclonals: evidence that an initially deposited species is A beta 42(43). Neuron. 13 [PubMed]
Kamenetz F et al. (2003). APP processing and synaptic function. Neuron. 37 [PubMed]
Kim DY et al. (2007). BACE1 regulates voltage-gated sodium channels and neuronal activity. Nature cell biology. 9 [PubMed]
Kloskowska E, Malkiewicz K, Winblad B, Benedikz E, Bruton JD. (2008). APPswe mutation increases the frequency of spontaneous Ca2+-oscillations in rat hippocampal neurons. Neuroscience letters. 436 [PubMed]
Knobloch M, Mansuy IM. (2008). Dendritic spine loss and synaptic alterations in Alzheimer's disease. Molecular neurobiology. 37 [PubMed]
Kuchibhotla KV, Lattarulo CR, Hyman BT, Bacskai BJ. (2009). Synchronous hyperactivity and intercellular calcium waves in astrocytes in Alzheimer mice. Science (New York, N.Y.). 323 [PubMed]
LaFerla FM, Green KN, Oddo S. (2007). Intracellular amyloid-beta in Alzheimer's disease. Nature reviews. Neuroscience. 8 [PubMed]
MacLean JN et al. (2005). Activity-independent coregulation of IA and Ih in rhythmically active neurons. Journal of neurophysiology. 94 [PubMed]
Manczak M et al. (2006). Mitochondria are a direct site of A beta accumulation in Alzheimer's disease neurons: implications for free radical generation and oxidative damage in disease progression. Human molecular genetics. 15 [PubMed]
Mattson MP, Chan SL. (2001). Dysregulation of cellular calcium homeostasis in Alzheimer's disease: bad genes and bad habits. Journal of molecular neuroscience : MN. 17 [PubMed]
Megías M, Emri Z, Freund TF, Gulyás AI. (2001). Total number and distribution of inhibitory and excitatory synapses on hippocampal CA1 pyramidal cells. Neuroscience. 102 [PubMed]
Migliore M. (2003). On the integration of subthreshold inputs from Perforant Path and Schaffer Collaterals in hippocampal CA1 pyramidal neurons. Journal of computational neuroscience. 14 [PubMed]
Migliore M, Novara G, Tegolo D. (2008). Single neuron binding properties and the magical number 7. Hippocampus. 18 [PubMed]
Migliore M, Shepherd GM. (2002). Emerging rules for the distributions of active dendritic conductances. Nature reviews. Neuroscience. 3 [PubMed]
Moreira PI, Santos MS, Moreno A, Oliveira C. (2001). Amyloid beta-peptide promotes permeability transition pore in brain mitochondria. Bioscience reports. 21 [PubMed]
Morse TM, Carnevale NT, Mutalik PG, Migliore M, Shepherd GM. (2010). Abnormal Excitability of Oblique Dendrites Implicated in Early Alzheimer's: A Computational Study. Frontiers in neural circuits. 4 [PubMed]
Nagy Z et al. (1995). Relative roles of plaques and tangles in the dementia of Alzheimer's disease: correlations using three sets of neuropathological criteria. Dementia (Basel, Switzerland). 6 [PubMed]
Palop JJ, Mucke L. (2010). Amyloid-beta-induced neuronal dysfunction in Alzheimer's disease: from synapses toward neural networks. Nature neuroscience. 13 [PubMed]
Parameshwaran K et al. (2007). Amyloid beta-peptide Abeta(1-42) but not Abeta(1-40) attenuates synaptic AMPA receptor function. Synapse (New York, N.Y.). 61 [PubMed]
Perez-Cruz C et al. (2011). Reduced spine density in specific regions of CA1 pyramidal neurons in two transgenic mouse models of Alzheimer's disease. The Journal of neuroscience : the official journal of the Society for Neuroscience. 31 [PubMed]
Plant LD et al. (2006). Amyloid beta peptide as a physiological modulator of neuronal 'A'-type K+ current. Neurobiology of aging. 27 [PubMed]
Ponomareva NV, Korovaitseva GI, Rogaev EI. (2008). EEG alterations in non-demented individuals related to apolipoprotein E genotype and to risk of Alzheimer disease. Neurobiology of aging. 29 [PubMed]
Reddy PH, Beal MF. (2008). Amyloid beta, mitochondrial dysfunction and synaptic damage: implications for cognitive decline in aging and Alzheimer's disease. Trends in molecular medicine. 14 [PubMed]
Rensink AA, de Waal RM, Kremer B, Verbeek MM. (2003). Pathogenesis of cerebral amyloid angiopathy. Brain research. Brain research reviews. 43 [PubMed]
Rowan M. (2012). Information-selectivity of Beta-amyloid pathology in an associative memory model. Frontiers in computational neuroscience. 6 [PubMed]
Rui Y et al. (2006). Acute effect of beta amyloid on synchronized spontaneous Ca2+ oscillations in cultured hippocampal networks. Cell biology international. 30 [PubMed]
Ruppin E, Reggia JA. (1995). A neural model of memory impairment in diffuse cerebral atrophy. The British journal of psychiatry : the journal of mental science. 166 [PubMed]
Sato S et al. (2008). Spatial learning impairment, enhanced CDK5/p35 activity, and downregulation of NMDA receptor expression in transgenic mice expressing tau-tubulin kinase 1. The Journal of neuroscience : the official journal of the Society for Neuroscience. 28 [PubMed]
Schenk D et al. (1999). Immunization with amyloid-beta attenuates Alzheimer-disease-like pathology in the PDAPP mouse. Nature. 400 [PubMed]
Selkoe DJ. (1999). Translating cell biology into therapeutic advances in Alzheimer's disease. Nature. 399 [PubMed]
Shankar GM et al. (2007). Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway. The Journal of neuroscience : the official journal of the Society for Neuroscience. 27 [PubMed]
Snyder EM et al. (2005). Regulation of NMDA receptor trafficking by amyloid-beta. Nature neuroscience. 8 [PubMed]
Terry RD. (2000). Cell death or synaptic loss in Alzheimer disease. Journal of neuropathology and experimental neurology. 59 [PubMed]
Terry RD et al. (1991). Physical basis of cognitive alterations in Alzheimer's disease: synapse loss is the major correlate of cognitive impairment. Annals of neurology. 30 [PubMed]
Texidó L, Martín-Satué M, Alberdi E, Solsona C, Matute C. (2011). Amyloid ß peptide oligomers directly activate NMDA receptors. Cell calcium. 49 [PubMed]
Wei W et al. (2010). Amyloid beta from axons and dendrites reduces local spine number and plasticity. Nature neuroscience. 13 [PubMed]
Zou X, Coyle D, Wong-Lin K, Maguire L. (2011). Computational study of hippocampal-septal theta rhythm changes due to ß-amyloid-altered ionic channels. PloS one. 6 [PubMed]
Migliore M, Tegolo D, Spera E, Unsworth N. (2016). On the cellular mechanisms underlying working memory capacity in humans Neural Network World. 4
Romani A et al. (2013). Computational modeling of the effects of amyloid-beta on release probability at hippocampal synapses. Frontiers in computational neuroscience. 7 [PubMed]