McKay BE, Molineux ML, Mehaffey WH, Turner RW. (2005). Kv1 K+ channels control Purkinje cell output to facilitate postsynaptic rebound discharge in deep cerebellar neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 25 [PubMed]

See more from authors: McKay BE · Molineux ML · Mehaffey WH · Turner RW

References and models cited by this paper
References and models that cite this paper

Akemann W, Knöpfel T. (2006). Interaction of Kv3 potassium channels and resurgent sodium current influences the rate of spontaneous firing of Purkinje neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 26 [PubMed]

Fernandez FR, Engbers JD, Turner RW. (2007). Firing dynamics of cerebellar purkinje cells. Journal of neurophysiology. 98 [PubMed]

Masoli S, Solinas S, D'Angelo E. (2015). Action potential processing in a detailed Purkinje cell model reveals a critical role for axonal compartmentalization. Frontiers in cellular neuroscience. 9 [PubMed]

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Molineux ML, Fernandez FR, Mehaffey WH, Turner RW. (2005). A-type and T-type currents interact to produce a novel spike latency-voltage relationship in cerebellar stellate cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. 25 [PubMed]

Ovsepian SV et al. (2013). A defined heteromeric KV1 channel stabilizes the intrinsic pacemaking and regulates the output of deep cerebellar nuclear neurons to thalamic targets. The Journal of physiology. 591 [PubMed]

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