Arman P et al. (2025). FGF14 Peptide Derivative Differentially Regulates Nav1.2 and Nav1.6 Function. Life (Basel, Switzerland). 15 [PubMed]

See more from authors: Arman P · Haghighijoo Z · Lupascu CA · Singh AK · Goode NA · Baumgartner TJ · Singh J · Xue Y · Wang P · Chen H · Antunes DA · Lijffijt M · Zhou J · Migliore M · Laezza F

References and models cited by this paper

Akin EJ et al. (2016). Single-Molecule Imaging of Nav1.6 on the Surface of Hippocampal Neurons Reveals Somatic Nanoclusters. Biophysical journal. 111 [PubMed]

Ben-Shalom R et al. (2017). Opposing Effects on NaV1.2 Function Underlie Differences Between SCN2A Variants Observed in Individuals With Autism Spectrum Disorder or Infantile Seizures. Biological psychiatry. 82 [PubMed]

Brackenbury WJ et al. (2010). Functional reciprocity between Na+ channel Nav1.6 and beta1 subunits in the coordinated regulation of excitability and neurite outgrowth. Proceedings of the National Academy of Sciences of the United States of America. 107 [PubMed]

Cha A, Ruben PC, George AL, Fujimoto E, Bezanilla F. (1999). Voltage sensors in domains III and IV, but not I and II, are immobilized by Na+ channel fast inactivation. Neuron. 22 [PubMed]

Cummins TR, Rush AM, Estacion M, Dib-Hajj SD, Waxman SG. (2009). Voltage-clamp and current-clamp recordings from mammalian DRG neurons. Nature protocols. 4 [PubMed]

Effraim PR et al. (2019). Fibroblast growth factor homologous factor 2 (FGF-13) associates with Nav1.7 in DRG neurons and alters its current properties in an isoform-dependent manner. Neurobiology of pain (Cambridge, Mass.). 6 [PubMed]

Gazina EV et al. (2015). 'Neonatal' Nav1.2 reduces neuronal excitability and affects seizure susceptibility and behaviour. Human molecular genetics. 24 [PubMed]

Goldfarb M et al. (2007). Fibroblast growth factor homologous factors control neuronal excitability through modulation of voltage-gated sodium channels. Neuron. 55 [PubMed]

Goldin AL. (2003). Mechanisms of sodium channel inactivation. Current opinion in neurobiology. 13 [PubMed]

Hines ML, Carnevale NT. (1997). The NEURON simulation environment. Neural computation. 9 [PubMed]

Hu W et al. (2009). Distinct contributions of Na(v)1.6 and Na(v)1.2 in action potential initiation and backpropagation. Nature neuroscience. 12 [PubMed]

Katz E et al. (2018). Role of sodium channel subtype in action potential generation by neocortical pyramidal neurons. Proceedings of the National Academy of Sciences of the United States of America. 115 [PubMed]

Laezza F et al. (2009). FGF14 N-terminal splice variants differentially modulate Nav1.2 and Nav1.6-encoded sodium channels. Molecular and cellular neurosciences. 42 [PubMed]

Liao Y et al. (2010). Molecular correlates of age-dependent seizures in an inherited neonatal-infantile epilepsy. Brain : a journal of neurology. 133 [PubMed]

Lou JY et al. (2005). Fibroblast growth factor 14 is an intracellular modulator of voltage-gated sodium channels. The Journal of physiology. 569 [PubMed]

Migliore R et al. (2018). The physiological variability of channel density in hippocampal CA1 pyramidal cells and interneurons explored using a unified data-driven modeling workflow PLOS Computational Biology. [PubMed]

Royeck M et al. (2008). Role of axonal NaV1.6 sodium channels in action potential initiation of CA1 pyramidal neurons. Journal of neurophysiology. 100 [PubMed]

Shavkunov AS et al. (2013). The fibroblast growth factor 14·voltage-gated sodium channel complex is a new target of glycogen synthase kinase 3 (GSK3). The Journal of biological chemistry. 288 [PubMed]

Spratt PWE et al. (2021). Paradoxical hyperexcitability from NaV1.2 sodium channel loss in neocortical pyramidal cells Cell reports. 36 [PubMed]

Spratt PWE et al. (2019). The Autism-Associated Gene Scn2a Contributes to Dendritic Excitability and Synaptic Function in the Prefrontal Cortex. Neuron. 103 [PubMed]

Tian C, Wang K, Ke W, Guo H, Shu Y. (2014). Molecular identity of axonal sodium channels in human cortical pyramidal cells. Frontiers in cellular neuroscience. 8 [PubMed]

Van Wart A, Trimmer JS, Matthews G. (2007). Polarized distribution of ion channels within microdomains of the axon initial segment. The Journal of comparative neurology. 500 [PubMed]

Xiao M, Bosch MK, Nerbonne JM, Ornitz DM. (2013). FGF14 localization and organization of the axon initial segment. Molecular and cellular neurosciences. 56 [PubMed]

Ye M et al. (2018). Differential roles of NaV1.2 and NaV1.6 in regulating neuronal excitability at febrile temperature and distinct contributions to febrile seizures. Scientific reports. 8 [PubMed]

Zhang J et al. (2021). Severe deficiency of the voltage-gated sodium channel NaV1.2 elevates neuronal excitability in adult mice. Cell reports. 36 [PubMed]

de Lera Ruiz M, Kraus RL. (2015). Voltage-Gated Sodium Channels: Structure, Function, Pharmacology, and Clinical Indications. Journal of medicinal chemistry. 58 [PubMed]

References and models that cite this paper
This website requires cookies and limited processing of your personal data in order to function. By continuing to browse or otherwise use this site, you are agreeing to this use. See our Privacy policy and how to cite and terms of use.