Akopian AN, Sivilotti L, Wood JN. (1996). A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons. Nature. 379 [PubMed]
Akopian AN et al. (1999). The tetrodotoxin-resistant sodium channel SNS has a specialized function in pain pathways. Nature neuroscience. 2 [PubMed]
Barkai O, Butterman R, Katz B, Lev S, Binshtok AM. (2020). The Input-Output Relation of Primary Nociceptive Neurons is Determined by the Morphology of the Peripheral Nociceptive Terminals. The Journal of neuroscience : the official journal of the Society for Neuroscience. 40 [PubMed]
Blair NT, Bean BP. (2002). Roles of tetrodotoxin (TTX)-sensitive Na+ current, TTX-resistant Na+ current, and Ca2+ current in the action potentials of nociceptive sensory neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 22 [PubMed]
Cox JJ et al. (2006). An SCN9A channelopathy causes congenital inability to experience pain. Nature. 444 [PubMed]
Cummins TR, Dib-Hajj SD, Waxman SG. (2004). Electrophysiological properties of mutant Nav1.7 sodium channels in a painful inherited neuropathy. The Journal of neuroscience : the official journal of the Society for Neuroscience. 24 [PubMed]
Cummins TR, Howe JR, Waxman SG. (1998). Slow closed-state inactivation: a novel mechanism underlying ramp currents in cells expressing the hNE/PN1 sodium channel. The Journal of neuroscience : the official journal of the Society for Neuroscience. 18 [PubMed]
Dover K, Solinas S, D'Angelo E, Goldfarb M. (2010). Long-term inactivation particle for voltage-gated sodium channels. The Journal of physiology. 588 [PubMed]
Goetz R et al. (2009). Crystal structure of a fibroblast growth factor homologous factor (FHF) defines a conserved surface on FHFs for binding and modulation of voltage-gated sodium channels. The Journal of biological chemistry. 284 [PubMed]
Goldfarb M et al. (2007). Fibroblast growth factor homologous factors control neuronal excitability through modulation of voltage-gated sodium channels. Neuron. 55 [PubMed]
Goldstein RH et al. (2019). Location and Plasticity of the Sodium Spike Initiation Zone in Nociceptive Terminals In Vivo. Neuron. 102 [PubMed]
Hines ML, Carnevale NT. (2001). NEURON: a tool for neuroscientists. The Neuroscientist : a review journal bringing neurobiology, neurology and psychiatry. 7 [PubMed]
Lou JY et al. (2005). Fibroblast growth factor 14 is an intracellular modulator of voltage-gated sodium channels. The Journal of physiology. 569 [PubMed]
Nassar MA et al. (2004). Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain. Proceedings of the National Academy of Sciences of the United States of America. 101 [PubMed]
Park DS et al. (2016). Fhf2 gene deletion causes temperature-sensitive cardiac conduction failure. Nature communications. 7 [PubMed]
Park DS et al. (2020). Ionic Mechanisms of Impulse Propagation Failure in the FHF2-Deficient Heart. Circulation research. 127 [PubMed]
Puranam RS et al. (2015). Disruption of Fgf13 causes synaptic excitatory-inhibitory imbalance and genetic epilepsy and febrile seizures plus. The Journal of neuroscience : the official journal of the Society for Neuroscience. 35 [PubMed]
Siekierska A et al. (2016). Gain-of-function FHF1 mutation causes early-onset epileptic encephalopathy with cerebellar atrophy. Neurology. 86 [PubMed]
Wang C, Chung BC, Yan H, Lee SY, Pitt GS. (2012). Crystal structure of the ternary complex of a NaV C-terminal domain, a fibroblast growth factor homologous factor, and calmodulin. Structure (London, England : 1993). 20 [PubMed]
Yang L et al. (2017). FGF13 Selectively Regulates Heat Nociception by Interacting with Nav1.7. Neuron. 93 [PubMed]
Yang Y et al. (2004). Mutations in SCN9A, encoding a sodium channel alpha subunit, in patients with primary erythermalgia. Journal of medical genetics. 41 [PubMed]