Alekov AK, Peter W, Mitrovic N, Lehmann-Horn F, Lerche H. (2001). Two mutations in the IV/S4-S5 segment of the human skeletal muscle Na+ channel disrupt fast and enhance slow inactivation. Neuroscience letters. 306 [PubMed]
Catterall WA. (2000). From ionic currents to molecular mechanisms: the structure and function of voltage-gated sodium channels. Neuron. 26 [PubMed]
Courtney MJ, Akerman KE, Coffey ET. (1997). Neurotrophins protect cultured cerebellar granule neurons against the early phase of cell death by a two-component mechanism. The Journal of neuroscience : the official journal of the Society for Neuroscience. 17 [PubMed]
D'Angelo E et al. (2001). Theta-frequency bursting and resonance in cerebellar granule cells: experimental evidence and modeling of a slow k+-dependent mechanism. The Journal of neuroscience : the official journal of the Society for Neuroscience. 21 [PubMed]
Diwakar S, Magistretti J, Goldfarb M, Naldi G, D'Angelo E. (2009). Axonal Na+ channels ensure fast spike activation and back-propagation in cerebellar granule cells. Journal of neurophysiology. 101 [PubMed]
Eaholtz G, Colvin A, Leonard D, Taylor C, Catterall WA. (1999). Block of brain sodium channels by peptide mimetics of the isoleucine, phenylalanine, and methionine (IFM) motif from the inactivation gate. The Journal of general physiology. 113 [PubMed]
Eaholtz G, Scheuer T, Catterall WA. (1994). Restoration of inactivation and block of open sodium channels by an inactivation gate peptide. Neuron. 12 [PubMed]
Featherstone DE, Richmond JE, Ruben PC. (1996). Interaction between fast and slow inactivation in Skm1 sodium channels. Biophysical journal. 71 [PubMed]
Frottin F et al. (2006). The proteomics of N-terminal methionine cleavage. Molecular & cellular proteomics : MCP. 5 [PubMed]
Goetz R et al. (2009). Crystal structure of a fibroblast growth factor homologous factor (FHF) defines a conserved surface on FHFs for binding and modulation of voltage-gated sodium channels. The Journal of biological chemistry. 284 [PubMed]
Goldfarb M. (2005). Fibroblast growth factor homologous factors: evolution, structure, and function And Function Cytokine Growth Factor Rev. 16
Goldfarb M et al. (2007). Fibroblast growth factor homologous factors control neuronal excitability through modulation of voltage-gated sodium channels. Neuron. 55 [PubMed]
Grieco TM, Malhotra JD, Chen C, Isom LL, Raman IM. (2005). Open-channel block by the cytoplasmic tail of sodium channel beta4 as a mechanism for resurgent sodium current. Neuron. 45 [PubMed]
Hartung H et al. (1997). Murine FGF-12 and FGF-13: expression in embryonic nervous system, connective tissue and heart. Mechanisms of development. 64 [PubMed]
Jung HY, Mickus T, Spruston N. (1997). Prolonged sodium channel inactivation contributes to dendritic action potential attenuation in hippocampal pyramidal neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 17 [PubMed]
Laezza F et al. (2009). FGF14 N-terminal splice variants differentially modulate Nav1.2 and Nav1.6-encoded sodium channels. Molecular and cellular neurosciences. 42 [PubMed]
Liu CJ, Dib-Hajj SD, Renganathan M, Cummins TR, Waxman SG. (2003). Modulation of the cardiac sodium channel Nav1.5 by fibroblast growth factor homologous factor 1B. The Journal of biological chemistry. 278 [PubMed]
Long SB, Campbell EB, Mackinnon R. (2005). Voltage sensor of Kv1.2: structural basis of electromechanical coupling. Science (New York, N.Y.). 309 [PubMed]
Lou JY et al. (2005). Fibroblast growth factor 14 is an intracellular modulator of voltage-gated sodium channels. The Journal of physiology. 569 [PubMed]
Pathak MM et al. (2007). Closing in on the resting state of the Shaker K(+) channel. Neuron. 56 [PubMed]
Patton DE, West JW, Catterall WA, Goldin AL. (1992). Amino acid residues required for fast Na(+)-channel inactivation: charge neutralizations and deletions in the III-IV linker. Proceedings of the National Academy of Sciences of the United States of America. 89 [PubMed]
Raman IM, Bean BP. (1997). Resurgent sodium current and action potential formation in dissociated cerebellar Purkinje neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 17 [PubMed]
Raman IM, Bean BP. (2001). Inactivation and recovery of sodium currents in cerebellar Purkinje neurons: evidence for two mechanisms. Biophysical journal. 80 [PubMed]
Remy S, Csicsvari J, Beck H. (2009). Activity-dependent control of neuronal output by local and global dendritic spike attenuation. Neuron. 61 [PubMed]
Rush AM et al. (2006). Differential modulation of sodium channel Na(v)1.6 by two members of the fibroblast growth factor homologous factor 2 subfamily. The European journal of neuroscience. 23 [PubMed]
Schoorlemmer J, Goldfarb M. (2001). Fibroblast growth factor homologous factors are intracellular signaling proteins. Current biology : CB. 11 [PubMed]
Shakkottai VG et al. (2009). FGF14 regulates the intrinsic excitability of cerebellar Purkinje neurons. Neurobiology of disease. 33 [PubMed]
Smallwood PM et al. (1996). Fibroblast growth factor (FGF) homologous factors: new members of the FGF family implicated in nervous system development. Proceedings of the National Academy of Sciences of the United States of America. 93 [PubMed]
Smith MR, Goldin AL. (1997). Interaction between the sodium channel inactivation linker and domain III S4-S5. Biophysical journal. 73 [PubMed]
Tucker K, Fadool DA. (2002). Neurotrophin modulation of voltage-gated potassium channels in rat through TrkB receptors is time and sensory experience dependent. The Journal of physiology. 542 [PubMed]
Ulbricht W. (2005). Sodium channel inactivation: molecular determinants and modulation. Physiological reviews. 85 [PubMed]
Wang Q, McEwen DG, Ornitz DM. (2000). Subcellular and developmental expression of alternatively spliced forms of fibroblast growth factor 14. Mechanisms of development. 90 [PubMed]
Wittmack EK et al. (2004). Fibroblast growth factor homologous factor 2B: association with Nav1.6 and selective colocalization at nodes of Ranvier of dorsal root axons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 24 [PubMed]
Diwakar S, Lombardo P, Solinas S, Naldi G, D'Angelo E. (2011). Local field potential modeling predicts dense activation in cerebellar granule cells clusters under LTP and LTD control. PloS one. 6 [PubMed]
Dover K et al. (2016). FHF-independent conduction of action potentials along the leak-resistant cerebellar granule cell axon. Nature communications. 7 [PubMed]
Marra C, Hartke TV, Ringkamp M, Goldfarb M. (2023). Enhanced sodium channel inactivation by temperature and FHF2 deficiency blocks heat nociception Pain. 164 [PubMed]
Upchurch CM et al. (2022). Long-Term Inactivation of Sodium Channels as a Mechanism of Adaptation in CA1 Pyramidal Cells The Journal of neuroscience : the official journal of the Society for Neuroscience. 42 [PubMed]