Badoual M et al. (2006). Biophysical and phenomenological models of multiple spike interactions in spike-timing dependent plasticity. International journal of neural systems. 16 [PubMed]

• Biophysical and phenomenological models of spike-timing dependent plasticity (Badoual et al. 2006) [Model]

Graupner M, Brunel N. (2007). STDP in a bistable synapse model based on CaMKII and associated signaling pathways. PLoS computational biology. 3 [PubMed]

• CaMKII system exhibiting bistability with respect to calcium (Graupner and Brunel 2007) [Model]

Kim M, Huang T, Abel T, Blackwell KT. (2010). Temporal sensitivity of protein kinase a activation in late-phase long term potentiation. PLoS computational biology. 6 [PubMed]

• Hippocampus CA1: Temporal sensitivity of signaling pathways underlying LTP (Kim et al. 2010) [Model]

Lindskog M, Kim M, Wikström MA, Blackwell KT, Kotaleski JH. (2006). Transient calcium and dopamine increase PKA activity and DARPP-32 phosphorylation. PLoS computational biology. 2 [PubMed]

• Model of DARPP-32 phosphorylation in striatal medium spiny neurons (Lindskog et al. 2006) [Model]

Manninen T, Hituri K, Kotaleski JH, Blackwell KT, Linne ML. (2010). Postsynaptic signal transduction models for long-term potentiation and depression. Frontiers in computational neuroscience. 4 [PubMed]

Mäki-Marttunen T, Iannella N, Edwards AG, Einevoll GT, Blackwell KT. (2020). A unified computational model for cortical post-synaptic plasticity. eLife. 9 [PubMed]

• Biochemically detailed model of LTP and LTD in a cortical spine (Maki-Marttunen et al 2020) [Model]

Nakano T, Doi T, Yoshimoto J, Doya K. (2010). A kinetic model of dopamine- and calcium-dependent striatal synaptic plasticity. PLoS computational biology. 6 [PubMed]

• A kinetic model of dopamine- and calcium-dependent striatal synaptic plasticity (Nakano et al. 2010) [Model]