Ashhad S, Narayanan R. (2013). Quantitative interactions between the A-type K+ current and inositol trisphosphate receptors regulate intraneuronal Ca2+ waves and synaptic plasticity. The Journal of physiology. 591 [PubMed]

• Calcium waves and mGluR-dependent synaptic plasticity in CA1 pyr. neurons (Ashhad & Narayanan 2013) [Model]

Diwakar S, Magistretti J, Goldfarb M, Naldi G, D'Angelo E. (2009). Axonal Na+ channels ensure fast spike activation and back-propagation in cerebellar granule cells. Journal of neurophysiology. 101 [PubMed]

• Multicompartmental cerebellar granule cell model (Diwakar et al. 2009) [Model]

Fineberg JD, Ritter DM, Covarrubias M. (2012). Modeling-independent elucidation of inactivation pathways in recombinant and native A-type Kv channels. The Journal of general physiology. 140 [PubMed]

• Neurophysiological impact of inactivation pathways in A-type K+ channels (Fineberg et al 2012) [Model]

Molineux ML, Fernandez FR, Mehaffey WH, Turner RW. (2005). A-type and T-type currents interact to produce a novel spike latency-voltage relationship in cerebellar stellate cells. The Journal of neuroscience : the official journal of the Society for Neuroscience. 25 [PubMed]

• IA and IT interact to set first spike latency (Molineux et al 2005) [Model]

Zheng Y et al. (2019). Deep Sequencing of Somatosensory Neurons Reveals Molecular Determinants of Intrinsic Physiological Properties Neuron.

• DRG neuron models investigate how ion channel levels regulate firing properties (Zheng et al 2019) [Model]