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• DCN fusiform cell (Ceballos et al. 2016) [Model]

Chemin J et al. (2002). Specific contribution of human T-type calcium channel isotypes (alpha(1G), alpha(1H) and alpha(1I)) to neuronal excitability. The Journal of physiology. 540 [PubMed]

Häusser M, Monsivais P. (2003). Less means more: inhibition of spontaneous firing triggers persistent increases in excitability. Neuron. 40 [PubMed]

Häusser M et al. (2004). The beat goes on: spontaneous firing in mammalian neuronal microcircuits. The Journal of neuroscience : the official journal of the Society for Neuroscience. 24 [PubMed]

Jaeger D et al. (2017). Robust Transmission of Rate Coding in the Inhibitory Purkinje Cell to Cerebellar Nuclei Pathway in Awake Mice PLOS Computational Biology.

• Robust transmission in the inhibitory Purkinje Cell to Cerebellar Nuclei pathway (Abbasi et al 2017) [Model]

Khaliq ZM, Gouwens NW, Raman IM. (2003). The contribution of resurgent sodium current to high-frequency firing in Purkinje neurons: an experimental and modeling study. The Journal of neuroscience : the official journal of the Society for Neuroscience. 23 [PubMed]

• Cerebellar Purkinje Cell: resurgent Na current and high frequency firing (Khaliq et al 2003) [Model]

Lin RJ, Jaeger D. (2011). Using computer simulations to determine the limitations of dynamic clamp stimuli applied at the soma in mimicking distributed conductance sources. Journal of neurophysiology. 105 [PubMed]

Magistretti J, Castelli L, Forti L, D'Angelo E. (2006). Kinetic and functional analysis of transient, persistent and resurgent sodium currents in rat cerebellar granule cells in situ: an electrophysiological and modelling study. The Journal of physiology. 573 [PubMed]

Mercer JN, Chan CS, Tkatch T, Held J, Surmeier DJ. (2007). Nav1.6 sodium channels are critical to pacemaking and fast spiking in globus pallidus neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 27 [PubMed]

• Nav1.6 sodium channel model in globus pallidus neurons (Mercer et al. 2007) [Model]

Ovsepian SV et al. (2013). A defined heteromeric KV1 channel stabilizes the intrinsic pacemaking and regulates the output of deep cerebellar nuclear neurons to thalamic targets. The Journal of physiology. 591 [PubMed]

• KV1 channel governs cerebellar output to thalamus (Ovsepian et al. 2013) [Model]

Paul JR et al. (2016). Regulation of persistent sodium currents by glycogen synthase kinase 3 encodes daily rhythms of neuronal excitability. Nature communications. 7 [PubMed]

• HH model neuron of the Suprachiasmatic Nucleus including a persistent Na+ channel (Paul et al 2016) [Model]

Steuber V, Jaeger D. (2013). Modeling the generation of output by the cerebellar nuclei. Neural networks : the official journal of the International Neural Network Society. 47 [PubMed]

• Robust transmission in the inhibitory Purkinje Cell to Cerebellar Nuclei pathway (Abbasi et al 2017) [Model]

Steuber V, Schultheiss NW, Silver RA, De Schutter E, Jaeger D. (2011). Determinants of synaptic integration and heterogeneity in rebound firing explored with data-driven models of deep cerebellar nucleus cells. Journal of computational neuroscience. 30 [PubMed]

• Cerebellar Nucleus Neuron (Steuber, Schultheiss, Silver, De Schutter & Jaeger, 2010) [Model]
• Robust transmission in the inhibitory Purkinje Cell to Cerebellar Nuclei pathway (Abbasi et al 2017) [Model]

Wetmore DZ, Mukamel EA, Schnitzer MJ. (2008). Lock-and-key mechanisms of cerebellar memory recall based on rebound currents. Journal of neurophysiology. 100 [PubMed]