Acker CD, White JA. (2007). Roles of IA and morphology in action potential propagation in CA1 pyramidal cell dendrites. Journal of computational neuroscience. 23 [PubMed]

• Roles of I(A) and morphology in AP prop. in CA1 pyramidal cell dendrites (Acker and White 2007) [Model]

Behabadi BF, Polsky A, Jadi M, Schiller J, Mel BW. (2012). Location-dependent excitatory synaptic interactions in pyramidal neuron dendrites. PLoS computational biology. 8 [PubMed]

• Excitatory synaptic interactions in pyramidal neuron dendrites (Behabadi et al. 2012) [Model]

Benuskova L, Abraham WC. (2007). STDP rule endowed with the BCM sliding threshold accounts for hippocampal heterosynaptic plasticity. Journal of computational neuroscience. 22 [PubMed]

Ebner C, Clopath C, Jedlicka P, Cuntz H. (2019). Unifying Long-Term Plasticity Rules for Excitatory Synapses by Modeling Dendrites of Cortical Pyramidal Neurons. Cell reports. 29 [PubMed]

• Four-pathway phenomenological synaptic plasticity model (Ebner et al. 2019) [Model]

Evans RC et al. (2012). The effects of NMDA subunit composition on calcium influx and spike timing-dependent plasticity in striatal medium spiny neurons. PLoS computational biology. 8 [PubMed]

• NMDA subunit effects on Calcium and STDP (Evans et al. 2012) [Model]

Graupner M, Brunel N. (2007). STDP in a bistable synapse model based on CaMKII and associated signaling pathways. PLoS computational biology. 3 [PubMed]

• CaMKII system exhibiting bistability with respect to calcium (Graupner and Brunel 2007) [Model]

Kobayashi T, Shimada Y, Fujiwara K, Ikeguchi T. (2017). Reproducing Infra-Slow Oscillations with Dopaminergic Modulation. Scientific reports. 7 [PubMed]

• Reproducing infra-slow oscillations with dopaminergic modulation (Kobayashi et al 2017) [Model]

Letzkus JJ, Kampa BM, Stuart GJ. (2006). Learning rules for spike timing-dependent plasticity depend on dendritic synapse location. The Journal of neuroscience : the official journal of the Society for Neuroscience. 26 [PubMed]

• STDP depends on dendritic synapse location (Letzkus et al. 2006) [Model]

Narayanan R, Chattarji S. (2010). Computational analysis of the impact of chronic stress on intrinsic and synaptic excitability in the hippocampus. Journal of neurophysiology. 103 [PubMed]

• Impact of dendritic atrophy on intrinsic and synaptic excitability (Narayanan & Chattarji, 2010) [Model]

Narayanan R, Johnston D. (2010). The h current is a candidate mechanism for regulating the sliding modification threshold in a BCM-like synaptic learning rule. Journal of neurophysiology. 104 [PubMed]

• BCM-like synaptic plasticity with conductance-based models (Narayanan Johnston, 2010) [Model]

Rabinowitch I, Segev I. (2006). The endurance and selectivity of spatial patterns of long-term potentiation/depression in dendrites under homeostatic synaptic plasticity. The Journal of neuroscience : the official journal of the Society for Neuroscience. 26 [PubMed]

• Homeostatic synaptic plasticity (Rabinowitch and Segev 2006a,b) [Model]

Tamosiunaite M, Porr B, Wörgötter F. (2007). Self-influencing synaptic plasticity: recurrent changes of synaptic weights can lead to specific functional properties. Journal of computational neuroscience. 23 [PubMed]

• Self-influencing synaptic plasticity (Tamosiunaite et al. 2007) [Model]

Toyoizumi T, Pfister JP, Aihara K, Gerstner W. (2007). Optimality model of unsupervised spike-timing-dependent plasticity: synaptic memory and weight distribution. Neural computation. 19 [PubMed]

Urakubo H, Honda M, Froemke RC, Kuroda S. (2008). Requirement of an allosteric kinetics of NMDA receptors for spike timing-dependent plasticity. The Journal of neuroscience : the official journal of the Society for Neuroscience. 28 [PubMed]

• An allosteric kinetics of NMDARs in STDP (Urakubo et al. 2008) [Model]

Yu Q, Tang H, Hu J, Tan KC. (2017). Precise-Spike-Driven Synaptic Plasticity for Hetero Association of Spatiotemporal Spike Patterns Neuromorphic Cognitive Systems: A Learning and Memory Centered Approach.

Zhou YD, Acker CD, Netoff TI, Sen K, White JA. (2005). Increasing Ca2+ transients by broadening postsynaptic action potentials enhances timing-dependent synaptic depression. Proceedings of the National Academy of Sciences of the United States of America. 102 [PubMed]