Akemann W, Knöpfel T. (2006). Interaction of Kv3 potassium channels and resurgent sodium current influences the rate of spontaneous firing of Purkinje neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 26 [PubMed]

• Cerebellar purkinje cell: interacting Kv3 and Na currents influence firing (Akemann, Knopfel 2006) [Model]

Chemin J et al. (2002). Specific contribution of human T-type calcium channel isotypes (alpha(1G), alpha(1H) and alpha(1I)) to neuronal excitability. The Journal of physiology. 540 [PubMed]

D'Angelo E et al. (2001). Theta-frequency bursting and resonance in cerebellar granule cells: experimental evidence and modeling of a slow k+-dependent mechanism. The Journal of neuroscience : the official journal of the Society for Neuroscience. 21 [PubMed]

• Bursting and resonance in cerebellar granule cells (D'Angelo et al. 2001) [Model]

Doiron B, Noonan L, Lemon N, Turner RW. (2003). Persistent Na+ current modifies burst discharge by regulating conditional backpropagation of dendritic spikes. Journal of neurophysiology. 89 [PubMed]

Dover K, Solinas S, D'Angelo E, Goldfarb M. (2010). Long-term inactivation particle for voltage-gated sodium channels. The Journal of physiology. 588 [PubMed]

Gurkiewicz M, Korngreen A, Waxman SG, Lampert A. (2011). Kinetic modeling of Nav1.7 provides insight into erythromelalgia-associated F1449V mutation. Journal of neurophysiology. 105 [PubMed]

• HMM of Nav1.7 WT and F1449V (Gurkiewicz et al. 2011) [Model]

Hallermann S, de Kock CP, Stuart GJ, Kole MH. (2012). State and location dependence of action potential metabolic cost in cortical pyramidal neurons. Nature neuroscience. 15 [PubMed]

• State and location dependence of action potential metabolic cost (Hallermann et al., 2012) [Model]

Häusser M, Monsivais P. (2003). Less means more: inhibition of spontaneous firing triggers persistent increases in excitability. Neuron. 40 [PubMed]

Häusser M et al. (2004). The beat goes on: spontaneous firing in mammalian neuronal microcircuits. The Journal of neuroscience : the official journal of the Society for Neuroscience. 24 [PubMed]

Khaliq ZM, Gouwens NW, Raman IM. (2003). The contribution of resurgent sodium current to high-frequency firing in Purkinje neurons: an experimental and modeling study. The Journal of neuroscience : the official journal of the Society for Neuroscience. 23 [PubMed]

• Cerebellar Purkinje Cell: resurgent Na current and high frequency firing (Khaliq et al 2003) [Model]

Kuo JJ, Lee RH, Zhang L, Heckman CJ. (2006). Essential role of the persistent sodium current in spike initiation during slowly rising inputs in mouse spinal neurones. The Journal of physiology. 574 [PubMed]

Liu CY, Xiao C, Fraser SE, Lester HA, Koos DS. (2012). Electrophysiological characterization of Grueneberg ganglion olfactory neurons: spontaneous firing, sodium conductance, and hyperpolarization-activated currents. Journal of neurophysiology. 108 [PubMed]

• Model of repetitive firing in Grueneberg ganglion olfactory neurons (Liu et al., 2012) [Model]

Magistretti J, Castelli L, Forti L, D'Angelo E. (2006). Kinetic and functional analysis of transient, persistent and resurgent sodium currents in rat cerebellar granule cells in situ: an electrophysiological and modelling study. The Journal of physiology. 573 [PubMed]

Mercer JN, Chan CS, Tkatch T, Held J, Surmeier DJ. (2007). Nav1.6 sodium channels are critical to pacemaking and fast spiking in globus pallidus neurons. The Journal of neuroscience : the official journal of the Society for Neuroscience. 27 [PubMed]

• Nav1.6 sodium channel model in globus pallidus neurons (Mercer et al. 2007) [Model]

Ohura S, Kamiya H. (2018). Sodium Channel-Dependent and -Independent Mechanisms Underlying Axonal Afterdepolarization at Mouse Hippocampal Mossy Fibers. eNeuro. 5 [PubMed]

• Evaluation of passive component of propagating AP in mossy fiber axons (Ohura & Kamiya 2018) [Model]

Raman IM, Bean BP. (2001). Inactivation and recovery of sodium currents in cerebellar Purkinje neurons: evidence for two mechanisms. Biophysical journal. 80 [PubMed]

• Cerebellar Purkinje Cell: resurgent Na current and high frequency firing (Khaliq et al 2003) [Model]

Royeck M et al. (2008). Role of axonal NaV1.6 sodium channels in action potential initiation of CA1 pyramidal neurons. Journal of neurophysiology. 100 [PubMed]

• Axonal NaV1.6 Sodium Channels in AP Initiation of CA1 Pyramidal Neurons (Royeck et al. 2008) [Model]

Solinas S et al. (2007). Computational reconstruction of pacemaking and intrinsic electroresponsiveness in cerebellar Golgi cells. Frontiers in cellular neuroscience. 1 [PubMed]

• Cerebellar Golgi cell (Solinas et al. 2007a, 2007b) [Model]

Venugopal S et al. (2019). Resurgent Na+ Current Offers Noise Modulation in Bursting Neurons. PLoS computational biology. 15 [PubMed]

• Resurgent Na+ current offers noise modulation in bursting neurons (Venugopal et al 2019) [Model]

Williams SR, Christensen SR, Stuart GJ, Häusser M. (2002). Membrane potential bistability is controlled by the hyperpolarization-activated current I(H) in rat cerebellar Purkinje neurons in vitro. The Journal of physiology. 539 [PubMed]

Zemel BM et al. (2021). Resurgent Na+ currents promote ultrafast spiking in projection neurons that drive fine motor control Nature communications. 12 [PubMed]

• Resurgent sodium transient current in zebra finch RA (Zemel et al., 2021) [Model]